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atherothrombosis
Items for "atherothrombosis"
The pharmacology of selective inhibition of COX-2
Selective inhibitors of cyclooxygenase (COX)-2 were developed to improve the safety of anti-inflammatory therapy in patients at elevated risk for gastrointestinal complications which are thought to be caused primarily by depression of COX-1 derived mucosal prostanoids...
Keywords: Clinical studies, atherothrombosis, atherosclerosis, inflammation, inflammatory mediators
10/2006 | Thrombosis and Haemostasis, SchattauerDoes a coxib-associated thrombotic risk limit the clinical use of the compounds as analgesic anti-inflammatory drugs? Arguments in favor
Non-steroidal anti-inflammatory agents (NSAIDs) and selective cyclooxygenase (COX-2) inhibitors (coxibs) are commonly used as analgesic and anti-inflammatory agents...
Keywords: acute myocardial infarction, coxib, atherothrombosis, cyclooxygenase-2, prothrombotic
10/2006 | Thrombosis and Haemostasis, SchattauerGene polymorphisms implicated in influencing susceptibility to venous and arterial thromboembolism: Frequency distribution in a healthy German population
Evolvement and progression of cardiovascular diseases affecting the venous and arterial system are influenced by a multitude of environmental and hereditary factors...
Keywords: Thrombophilia, Venous thromboembolism, atherothrombosis, SNP
10/2006 | Thrombosis and Haemostasis, SchattauerThe F11 receptor (F11R/JAM-A) in atherothrombosis: Overexpression of F11R in atherosclerotic plaques
F11R is the gene name for an adhesion protein, called the F11-receptor, aka JAM-A, which under normal physiological conditions is expressed constitutively on the surface of platelets and localized within tight junctions of endothelial cells (EC)...
Keywords: F11R, F11 receptor, F11R/JAM-A, inflammatory thrombosis, atherosclerosis, atherothrombosis
00/0000 | Thrombosis and Haemostasis, Schattauer